ABOP Orofacial Pain study materials
ABOP Orofacial Pain study resources

ABOP Orofacial Pain common mistakes

ABOP Orofacial Pain · Understand the traps and how to avoid them.

Mistake 1

Assigning a single diagnosis to a patient whose burning mouth symptoms began after starting an SSRI and who also has mild depressive symptoms, rather than building a differential that separates secondary causes from primary burning mouth syndrome.

Candidates anchor on the most familiar label (BMS) once they hear 'burning mouth' and stop the diagnostic workup, because the term feels like a diagnosis rather than a diagnosis of exclusion.

What to do instead

Before committing to primary BMS, explicitly list and rule out local (candidiasis, lichen planus, xerostomia, galvanic), systemic (B12/folate/iron deficiency, diabetes, hypothyroidism), and medication-related causes, and document which were excluded and how.

Mistake 2

Treating a unilateral throbbing headache with conjunctival injection, lacrimation, and ptosis as trigeminal neuralgia because the pain is in the V1 distribution.

Both conditions are unilateral and severe, and candidates pattern-match on 'facial pain in trigeminal territory' without applying the temporal and autonomic criteria that separate them.

What to do instead

For any severe unilateral facial/periorbital pain, actively test the trigeminal autonomic cephalalgia criteria (attack duration, autonomic signs, restlessness) versus the TN criteria (brief electric-shock paroxysms, trigger zones, no autonomic features) before diagnosing.

Mistake 3

Interpreting a positive response to a diagnostic local anesthetic injection as proof of a specific peripheral pain generator without a control injection or concordant history.

Candidates treat 'the block worked' as definitive when a placebo/expectancy effect and spread to adjacent structures can produce the same relief, and the block's target anatomy is often not verified.

What to do instead

Pair every diagnostic block with a documented pre-block pain score, a short-acting versus long-acting comparison or saline control when feasible, and a pre-specified threshold for what magnitude of relief counts as positive.

Mistake 4

Recommending irreversible occlusal adjustment or full-coverage restoration as first-line therapy for a patient with masticatory muscle pain and no degenerative joint disease.

The historical occlusal-equilibrium paradigm persists in training and in the referral network, and candidates want to 'fix the bite' they can see rather than address a biopsychosocial pain condition.

What to do instead

Default to conservative, reversible, evidence-based TMD management (education, self-management, physical therapy, cognitive behavioral approaches, short-term pharmacotherapy) and reserve irreversible occlusal therapy for clearly defined structural indications documented in the record.

Mistake 5

Failing to screen for sleep-disordered breathing before prescribing a muscle relaxant, benzodiazepine, or opioid to a patient with chronic orofacial pain and daytime somnolence.

Candidates compartmentalize the pain visit and the sleep visit and do not connect sedating analgesics to undiagnosed obstructive sleep apnea risk.

What to do instead

Include a validated sleep apnea screen (e.g., STOP-BANG) and a medication-sedation interaction check in every chronic orofacial pain intake, and document the result before any sedating prescription.

Mistake 6

Diagnosing trigeminal neuralgia in a patient with continuous, non-paroxysmal facial pain and normal neurologic exam because the pain is in the trigeminal distribution.

The name 'trigeminal' invites overapplication, and candidates underweight the requirement for brief, shock-like, electrically paroxysmal pain with a refractory period.

What to do instead

Apply the formal diagnostic criteria explicitly: paroxysmal electric-shock quality, duration of seconds, triggerable by innocuous stimuli, with a documented neurologic exam; if continuous pain predominates, consider painful posttraumatic trigeminal neuropathy or persistent idiopathic facial pain instead.

Mistake 7

Ordering MRI or CT as the first step for a patient with a clear myogenous TMD presentation and no red flags, then treating the normal image as reassurance that no treatment is needed.

Candidates equate thoroughness with imaging and are uncomfortable diagnosing a functional pain condition clinically, so imaging substitutes for a structured history and exam.

What to do instead

Complete a structured clinical exam (palpation, range of motion, joint sounds, loading tests, red-flag screen) first; reserve imaging for red flags, suspected structural pathology, or failed conservative care, and document the clinical reasoning either way.

Mistake 8

Repeatedly escalating opioid dose in a patient with chronic orofacial pain who reports functional improvement but shows no change in pain score, without reassessing the functional endpoint.

Candidates treat pain intensity as the sole outcome measure and interpret stable pain scores as treatment failure, driving dose escalation past the point of benefit.

What to do instead

Define functional goals (sleep, chewing, work, activity) at baseline and titrate or taper based on function and adverse effects, not pain score alone; document an exit strategy and monitoring plan at initiation.

Mistake 9

Missing the diagnosis of giant cell arteritis in an older patient with new temporal headache and jaw claudication because the presentation is attributed to TMD.

TMD is the most common orofacial pain diagnosis in the clinic, so candidates anchor on it and do not apply an age-based red-flag screen for secondary headache.

What to do instead

For any new headache in a patient over 50, explicitly ask about jaw claudication, scalp tenderness, and visual symptoms, check ESR/CRP, and treat as an urgent referral rather than a TMD trial.

Mistake 10

Building a treatment plan entirely around peripheral nociception for a patient with chronic pain and high catastrophizing, without addressing central sensitization or psychosocial modifiers.

Candidates trained in structural models default to tissue-based interventions and are uncomfortable raising psychological factors, so the plan omits the mechanisms most likely maintaining the pain.

What to do instead

Include a brief psychosocial screen (e.g., pain catastrophizing, fear-avoidance, depression/anxiety, sleep) in the assessment and build a multidisciplinary plan with graded activity and cognitive behavioral components when central or psychosocial contributors are identified.