ABS Surgical Critical Care study materials
ABS Surgical Critical Care study resources

ABS Surgical Critical Care cheat sheet

ABS Surgical Critical Care · Keep the key ideas within reach.

Quick reference

Biology of Critical Illness and Organ System Support

  • Oxygen delivery: DO2 = CO x CaO2 x 10; CaO2 = (1.34 x Hb x SaO2) + (0.0031 x PaO2).

  • Oxygen consumption: VO2 = CO x (CaO2 - CvO2) x 10; normal VO2 is ~110-160 mL/min/m2.

  • Shock is defined by inadequate DO2 relative to VO2; lactate rise and widened ScvO2-SvO2 gap signal dysoxia.

  • Mixed venous saturation (SvO2) <65% or ScvO2 <70% suggests inadequate cardiac output or anemia.

  • Acute respiratory distress syndrome (Berlin): PaO2/FiO2 <=300 with PEEP >=5, bilateral opacities, onset within 1 week, not cardiogenic.

  • Lung-protective ventilation: tidal volume 6 mL/kg predicted body weight, plateau pressure <=30 cmH2O.

  • Acute kidney injury (KDIGO): stage 1 = creatinine rise >=0.3 mg/dL or 1.5-1.9x baseline; stage 3 = >=3x or creatinine >=4.0 or RRT.

  • Continuous renal replacement therapy is preferred in hemodynamically unstable patients; intermittent HD for stable patients.

  • Sepsis-3: suspected infection plus SOFA increase >=2; septic shock = vasopressor need for MAP >=65 plus lactate >2 mmol/L despite fluids.

Quick reference

Physiologic Responses to Trauma, Burns, and Inflammation

  • Cushing reflex: hypertension with widened pulse pressure and bradycardia indicates elevated intracranial pressure.

  • Beck triad of cardiac tamponade: hypotension, distended neck veins, muffled heart sounds.

  • Burn fluid resuscitation (Parkland): 4 mL x body weight (kg) x %TBSA; half in first 8 hours from injury time.

  • Rule of Nines: each arm 9%, each leg 18%, anterior trunk 18%, posterior trunk 18%, head 9%, perineum 1%.

  • Systemic inflammatory response syndrome criteria: temperature >38 or <36 C, HR >90, RR >20 or PaCO2 <32, WBC >12k or <4k or >10% bands.

  • Compensatory anti-inflammatory response follows SIRS; combined state is mixed antagonist response syndrome.

  • Trauma triad of death: hypothermia, acidosis, and coagulopathy; each worsens the others.

  • Hypermetabolic burn response: elevated resting energy expenditure up to 2x baseline, increased catecholamines and cortisol.

  • Abdominal compartment syndrome: intra-abdominal pressure >20 mmHg with new organ dysfunction; decompress with laparotomy.

Quick reference

Comorbidities and Complications Unique to Surgical Patients

  • Revised Cardiac Risk Index predictors: ischemic heart disease, heart failure, cerebrovascular disease, insulin-dependent diabetes, creatinine >2.0, high-risk surgery.

  • Postoperative atrial fibrillation after noncardiac surgery: rate control first; anticoagulation decided by CHA2DS2-VASc and bleeding risk.

  • Venous thromboembolism prophylaxis: Caprini score guides modality; pharmacologic prophylaxis for moderate-high risk unless contraindicated.

  • Delirium risk factors: age >65, cognitive impairment, sensory deprivation, sleep disruption, immobility, pain, polypharmacy.

  • Anastomotic leak presents with tachycardia, fever, ileus, and rising lactate; CT with contrast is diagnostic.

  • Postoperative ileus vs. mechanical obstruction: ileus has diffuse air-fluid levels and no transition point; obstruction shows a transition zone.

  • Acute mesenteric ischemia: pain out of proportion to exam; lactate and metabolic acidosis are late findings; CT angiography is diagnostic.

  • Malignant hyperthermia: hypercarbia, rigidity, hyperthermia; treat with dantrolene 2.5 mg/kg IV and stop triggering agents.

  • Alcohol withdrawal: CIWA-Ar guides therapy; benzodiazepines are first-line; thiamine before glucose in malnourished patients.

Quick reference

Nonoperative Supportive Care Techniques

  • Mechanical ventilation modes: volume-control delivers set tidal volume; pressure-control sets inspiratory pressure; both limit barotrauma if plateau <=30.

  • PEEP improves oxygenation by recruiting alveoli and reducing shunt; excessive PEEP causes barotrauma and hypotension.

  • Prone positioning for ARDS: 12-16 hours/day improves mortality when PaO2/FiO2 <150.

  • Vasopressors: norepinephrine first-line for septic shock; add vasopressin or epinephrine if MAP remains <65.

  • Fluid resuscitation: balanced crystalloids preferred over normal saline to reduce hyperchloremic acidosis.

  • Enteral nutrition: start within 24-48 hours in critically ill patients; trophic feeds acceptable if not tolerating full feeds.

  • Sedation: target RASS -2 to 0; daily interruption reduces ventilator days and ICU length of stay.

  • Glycemic control: target 140-180 mg/dL in critically ill patients; avoid hypoglycemia.

  • Stress ulcer prophylaxis: indicated with mechanical ventilation >48h or coagulopathy; PPI or H2 blocker.

Quick reference

Critical Illness and Surgical Procedures, Leadership and Administration

  • Damage control surgery: abbreviated laparotomy, temporary closure, ICU resuscitation, then planned re-exploration.

  • Timing of surgery in septic shock: source control as soon as feasible; delay only for hemodynamic stabilization with ongoing vasopressors.

  • Perioperative risk: emergency surgery carries higher mortality than elective; ASA class and functional capacity refine risk.

  • ICU admission criteria: need for vasopressors, mechanical ventilation, invasive monitoring, or continuous renal replacement.

  • ICU staffing models: intensivist-led high-intensity staffing reduces mortality and length of stay versus low-intensity models.

  • Rapid response systems: early warning scores trigger evaluation; reduce cardiac arrests outside ICU.

  • Family-centered rounds and structured communication (e.g., SBAR) improve handoffs and reduce adverse events.

  • Quality metrics: ventilator-associated events, central line-associated bloodstream infections, catheter-associated UTIs, and delirium prevalence are tracked.

  • Triage and resource allocation: SOFA score and daily reassessment guide ICU bed allocation during surge.