WCC study materials
WCC study resources

WCC flashcards

WCC · Build recall with a focused review session.

32 cards. Read the term and the definition here. The deck at the top of this page is optional.

Epidermis
Avascular outer skin layer that receives oxygen by diffusion and provides the primary barrier; keratinocytes migrate from the basal layer during epithelialization.
Hemostasis phase
Immediate wound healing phase with vasoconstriction, platelet aggregation, and fibrin clot formation to stop bleeding and provide a provisional matrix.
Inflammatory phase
Early healing phase, usually days 0 to 4, marked by neutrophils, macrophages, and debridement of bacteria and debris.
Proliferative phase
Healing phase from about day 3 to 3 weeks involving fibroblast collagen deposition, angiogenesis, granulation tissue, wound contraction, and epithelialization.
Wound area formula
For simple wounds, area equals longest length times perpendicular width in centimeters, reported as square centimeters; irregular wounds require planimetry.
Undermining measurement
Measured in centimeters using a clock face with 12 o'clock at the patient's head; record extent from the wound edge under intact skin.
PUSH tool
Pressure Ulcer Scale for Healing scores wound area, exudate amount, and tissue type; total 0 to 17, with higher scores indicating worse healing.
Braden Scale
Six-item pressure injury risk tool scoring 6 to 23; lower scores indicate higher risk, and scores of 18 or less often trigger prevention.
Stage 1 pressure injury
Intact skin with non-blanchable erythema; in darker skin, color change may not blanch, but skin remains intact without open wound.
Stage 3 pressure injury
Full-thickness skin loss with adipose visible; fascia, muscle, tendon, cartilage, and bone are not exposed; slough or eschar may be present.
Deep tissue pressure injury
Intact or non-intact skin with persistent deep maroon or purple discoloration or blood-filled blister from pressure and shear damage to underlying tissue.
Unstageable pressure injury
Full-thickness pressure injury obscured by slough or eschar; true depth cannot be determined until enough necrotic tissue is removed to visualize wound base.
Venous ulcer features
Usually shallow, irregular, moist, and located near the medial malleolus, with edema, hemosiderin staining, and lipodermatosclerosis; compression is key therapy.
Ankle-brachial index
Ratio of ankle systolic pressure to brachial systolic pressure; values below 0.8 suggest arterial disease and compression must be used cautiously or avoided.
Arterial ulcer features
Punched-out, dry, pale, painful wounds often on distal or lateral legs, with weak pulses, hair loss, and shiny skin; compression is contraindicated if ischemia is severe.
Lymphatic ulcer
Ulcer associated with chronic lymphedema, often with hyperkeratosis, papillomatosis, and lymphorrhea; managed with decongestive therapy and compression when arterial flow allows.
Monofilament test
10-g monofilament examination assesses loss of protective sensation in the foot; missed points indicate neuropathy and increased ulcer risk.
Total contact cast
Irremovable offloading device for plantar neuropathic ulcers that redistributes pressure and improves healing more than removable walkers.
Charcot foot
Neuropathic complication with a warm, red, swollen foot and possible deformity; requires immediate offloading and referral to prevent collapse.
Probe-to-bone test
Blunt sterile probe gently explores a diabetic foot ulcer; bone contact raises suspicion for osteomyelitis and warrants imaging and specialist evaluation.
TIME framework
Wound bed preparation model: T issue nonviable, Infection or inflammation, Moisture imbalance, and Edge nonadvancing; guides debridement and dressing selection.
Autolytic debridement
Selective, slow debridement using moisture-retentive dressings that let endogenous enzymes liquefy necrotic tissue; requires adequate perfusion and infection control.
Sharp debridement
Selective removal of necrotic tissue with sterile instruments; contraindicated in unassessed arterial insufficiency, coagulopathy, or ischemic tissue without vascular evaluation.
Biofilm
Polymicrobial community embedded in extracellular matrix that adheres to wound surfaces, resists antibiotics, and requires mechanical disruption plus antimicrobial therapy.
Critical colonization
Intermediate bioburden state with delayed healing, increased exudate, or new pain but without classic systemic infection signs; requires topical antimicrobial and debridement.
Wound culture method
For chronic wounds, obtain tissue biopsy or deep swab after cleansing rather than superficial swab; culture only when clinical infection is present.
Hydrogel dressing
Water-based topical dressing that rehydrates dry wounds and supports autolytic debridement; may macerate surrounding skin if exudate is moderate to high.
Alginate dressing
Highly absorbent dressing derived from seaweed that forms a gel in exudate; used for moderate to high exudate with a secondary cover, not dry wounds.
Silver dressing
Antimicrobial dressing for infected or critically colonized wounds; use for limited duration with reassessment because prolonged use may impair healing and select resistance.
Protein needs in wounds
Adults with pressure injuries and malnutrition often need 1.25 to 1.5 grams of protein per kilogram daily, with 30 to 35 kcal/kg/day.
Teach-back method
Education technique where the patient explains information in their own words; confirms understanding of offloading, skin checks, and warning signs.
Premedication for dressing
Give prescribed analgesia before painful dressing changes; timing depends on onset, and nonadherent dressings reduce trauma and pain.